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dc.contributor.author | Klionsky D.J. | |
dc.contributor.author | Petroni G. | |
dc.contributor.author | Amaravadi R.K. | |
dc.contributor.author | Baehrecke E.H. | |
dc.contributor.author | Ballabio A. | |
dc.contributor.author | Boya P. | |
dc.contributor.author | Bravo-San Pedro J.M. | |
dc.contributor.author | Cadwell K. | |
dc.contributor.author | Cecconi F. | |
dc.contributor.author | Choi A.M.K. | |
dc.contributor.author | Choi M.E. | |
dc.contributor.author | Chu C.T. | |
dc.contributor.author | Codogno P. | |
dc.contributor.author | Colombo M. | |
dc.contributor.author | Cuervo A.M. | |
dc.contributor.author | Deretic V. | |
dc.contributor.author | Dikic I. | |
dc.contributor.author | Elazar Z. | |
dc.contributor.author | Eskelinen E.L. | |
dc.contributor.author | Fimia G.M. | |
dc.contributor.author | Gewirtz D.A. | |
dc.contributor.author | Green D.R. | |
dc.contributor.author | Hansen M. | |
dc.contributor.author | Jäättelä M. | |
dc.contributor.author | Johansen T. | |
dc.contributor.author | Juhász G. | |
dc.contributor.author | Karantza V. | |
dc.contributor.author | Kraft C. | |
dc.contributor.author | Kroemer G. | |
dc.contributor.author | Ktistakis N.T. | |
dc.contributor.author | Kumar S. | |
dc.contributor.author | Lopez-Otin C. | |
dc.contributor.author | Macleod K.F. | |
dc.contributor.author | Madeo F. | |
dc.contributor.author | Martinez J. | |
dc.contributor.author | Meléndez A. | |
dc.contributor.author | Mizushima N. | |
dc.contributor.author | Münz C. | |
dc.contributor.author | Penninger J.M. | |
dc.contributor.author | Perera R. | |
dc.contributor.author | Piacentini M. | |
dc.contributor.author | Reggiori F. | |
dc.contributor.author | Rubinsztein D.C. | |
dc.contributor.author | Ryan K. | |
dc.contributor.author | Sadoshima J. | |
dc.contributor.author | Santambrogio L. | |
dc.contributor.author | Scorrano L. | |
dc.contributor.author | Simon H.U. | |
dc.contributor.author | Simon A.K. | |
dc.contributor.author | Simonsen A. | |
dc.contributor.author | Stolz A. | |
dc.contributor.author | Tavernarakis N. | |
dc.contributor.author | Tooze S.A. | |
dc.contributor.author | Yoshimori T. | |
dc.contributor.author | Yuan J. | |
dc.contributor.author | Yue Z. | |
dc.contributor.author | Zhong Q. | |
dc.contributor.author | Galluzzi L. | |
dc.contributor.author | Pietrocola F. | |
dc.date.accessioned | 2022-02-09T20:33:25Z | |
dc.date.available | 2022-02-09T20:33:25Z | |
dc.date.issued | 2021 | |
dc.identifier.issn | 0261-4189 | |
dc.identifier.uri | https://dspace.kpfu.ru/xmlui/handle/net/168988 | |
dc.description.abstract | Autophagy is a core molecular pathway for the preservation of cellular and organismal homeostasis. Pharmacological and genetic interventions impairing autophagy responses promote or aggravate disease in a plethora of experimental models. Consistently, mutations in autophagy-related processes cause severe human pathologies. Here, we review and discuss preclinical data linking autophagy dysfunction to the pathogenesis of major human disorders including cancer as well as cardiovascular, neurodegenerative, metabolic, pulmonary, renal, infectious, musculoskeletal, and ocular disorders. | |
dc.relation.ispartofseries | EMBO Journal | |
dc.subject | aging | |
dc.subject | cancer | |
dc.subject | inflammation | |
dc.subject | metabolic syndromes | |
dc.subject | neurodegeneration | |
dc.title | Autophagy in major human diseases | |
dc.type | Review | |
dc.relation.ispartofseries-issue | 19 | |
dc.relation.ispartofseries-volume | 40 | |
dc.collection | Публикации сотрудников КФУ | |
dc.source.id | SCOPUS02614189-2021-40-19-SID85113807620 |