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Browsing by Author "Baumer Y."

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CD98 regulates vascular smooth muscle cell proliferation in atherosclerosis

Baumer Y.; McCurdy S.; Alcala M.; Mehta N.; Lee B.; Ginsberg M.; Boisvert W. (2017)

© 2016 Elsevier Ireland LtdBackground and aims Vascular smooth muscle cells (VSMC) migrate and proliferate to form a stabilizing fibrous cap that encapsulates atherosclerotic plaques. CD98 is a transmembrane protein made ...
Cluster of differentiation 43 deficiency in leukocytes leads to reduced atherosclerosis - Brief report

Meiler S.; Baumer Y.; McCurdy S.; Lee B.; Kitamoto S.; Boisvert W. (2016)

© 2014 American Heart Association, Inc.OBJECTIVE - : The aim of this study was to investigate the role of cluster of differentiation 43 (CD43), an integral membrane glycoprotein with both proadhesive and antiadhesive ...
Cluster of differentiation 43 deficiency in leukocytes leads to reduced atherosclerosis - Brief report

Meiler S.; Baumer Y.; McCurdy S.; Lee B.; Kitamoto S.; Boisvert W. (2015)

© 2014 American Heart Association, Inc. OBJECTIVE - : The aim of this study was to investigate the role of cluster of differentiation 43 (CD43), an integral membrane glycoprotein with both proadhesive and antiadhesive ...
Hyperlipidemia-induced cholesterol crystal production by endothelial cells promotes atherogenesis

Baumer Y.; McCurdy S.; Weatherby T.; Mehta N.; Halbherr S.; Halbherr P.; Yamazaki N.; Boisvert W. (2017)

© 2017 The Author(s). Endothelial cells (EC) play a key role in atherosclerosis. Although EC are in constant contact with low density lipoproteins (LDL), how EC process LDL and whether this influences atherogenesis, is ...
Macrophage-specific expression of IL-37 in hyperlipidemic mice attenuates atherosclerosis

McCurdy S.; Baumer Y.; Toulmin E.; Lee B.; Boisvert W. (2017)

Copyright © 2017 by The American Association of Immunologists, Inc. inflammation, as well as the formation of lipid-laden macrophage foam cells within the vessel wall. IL-37 is recognized as an important anti-inflammatory ...
MicroRNA 302a is a novel modulator of cholesterol homeostasis and atherosclerosis

Meiler S.; Baumer Y.; Toulmin E.; Seng K.; Boisvert W. (2016)

© 2014 American Heart Association, Inc.OBJECTIVE - : Macrophage foam cell formation is a key feature of atherosclerosis. Recent studies have shown that specific microRNAs (miRs) are regulated in modified low-density ...
MicroRNA 302a is a novel modulator of cholesterol homeostasis and atherosclerosis

Meiler S.; Baumer Y.; Toulmin E.; Seng K.; Boisvert W. (2015)

© 2014 American Heart Association, Inc. OBJECTIVE - : Macrophage foam cell formation is a key feature of atherosclerosis. Recent studies have shown that specific microRNAs (miRs) are regulated in modified low-density ...
Regulation of monocyte/macrophage polarisation by extracellular RNA

Cabrera-Fuentes H.; Lopez M.; McCurdy S.; Fischer S.; Meiler S.; Baumer Y.; Galuska S.; Preissner K.; Boisvert W. (2015)

© Schattauer 2015. Monocytes/macrophages respond to external stimuli with rapid changes in the expression of numerous inflammation-related genes to undergo polarisation towards the M1 (pro-inflammatory) or M2 (antiinflammatory) ...
Role of extracellular rna in atherosclerotic plaque formation in mice

Simsekyilmaz S.; Cabrera-Fuentes H.; Meiler S.; Kostin S.; Baumer Y.; Liehn E.; Weber C.; Boisvert W.; Preissner K.; Zernecke A. (2014)

BACKGROUND -: Atherosclerosis and vascular remodeling after injury are driven by inflammation and mononuclear cell infiltration. Extracellular RNA (eRNA) has recently been implicated to become enriched at sites of tissue ...
Ultramorphological analysis of plaque advancement and cholesterol crystal formation in Ldlr knockout mouse atherosclerosis

Baumer Y.; McCurdy S.; Jin X.; Weatherby T.; Dey A.; Mehta N.; Yap J.; Kruth H.; Boisvert W. (2019)

© 2019 Elsevier B.V. Backgound and aims: The low-density lipoprotein receptor-deficient (Ldlr−/−) mouse has been utilized by cardiovascular researchers for more than two decades to study atherosclerosis. However, there has ...

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Browsing by Author "Baumer Y."

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